Peptides studied across the aging axis — senescence, NAD+, and resilience.
Longevity research examines peptides that act on the hallmarks of aging: cellular senescence, mitochondrial decline, telomere maintenance, NAD+ metabolism, and the gradual loss of circadian and hormonal regulation. The goal in most models is healthspan — preserving function — rather than a single disease endpoint.
Compounds studied here often originate as endogenous regulatory peptides whose expression falls with age. Because aging endpoints are slow and multifactorial, this area leans heavily on biomarker studies, model organisms, and mechanistic work rather than large outcome trials.
Mitochondrially-encoded peptide with reported insulin-sensitizing activity.
View profileSynthetic tetrapeptide investigated for telomerase activity and circadian effects.
View profileSmall-molecule NNMT inhibitor (often catalogued alongside peptides).
View profileEssential redox cofactor central to mitochondrial bioenergetics and sirtuin activity.
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